Heat shock (41°C, 1 h) inhibits both Rac and Cdc 42 activation and
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A systematic, cell-based comparison of G-protein binding domains for their potential as localization-based, genetically encoded biosensors of Cdc42 or Rac activity
Heat shock (41°C, 1 h) inhibits both Rac and Cdc 42 activation and
Cdc42 deficiency induces podocyte apoptosis by inhibiting the Nwasp/stress fibers/YAP pathway
The heat-shock, or HSF1-mediated proteotoxic stress, response in cancer: from proteomic stability to oncogenesis
Reversal of phenotypes of cellular senescence by pan-mTOR inhibition
The role of lipid metabolic reprogramming in tumor microenvironment
Active and Inactive Cdc42 Differ in Their Insert Region Conformational Dynamics. - Abstract - Europe PMC
Activation of Cdc42, Rac, PAK, and Rho-Kinase in Response to Hepatocyte Growth Factor Differentially Regulates Epithelial Cell Colony Spreading and Dissociation
Full article: Cdc42 and Phosphoinositide 3-Kinase Drive Rac-Mediated Actin Polymerization Downstream of c-Met in Distinct and Common Pathways
Zz c0210-1002 hsp-us_lowres
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